Ethyl pyruvate does not require microglia for mediating neuroprotection after excitotoxic injury

AIMS: Ethyl pyruvate (EP) mediates protective effects after neuronal injury. Besides a direct conservation of damaged neurons, the modulation of indigenous glial cells has been suggested as one important mechanism for EP‐related neuroprotection. However, the specific contribution of glial cells is s...

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Detaylı Bibliyografya
Yayımlandı:CNS Neurosci Ther
Asıl Yazarlar: Pieroh, Philipp, Wagner, Daniel‐Christoph, Ghadban, Chalid, Birkenmeier, Gerd, Dehghani, Faramarz
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: John Wiley and Sons Inc. 2017
Konular:
Original Articles
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC6492706/
https://ncbi.nlm.nih.gov/pubmed/28836378
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/cns.12725
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