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Ethyl pyruvate does not require microglia for mediating neuroprotection after excitotoxic injury
AIMS: Ethyl pyruvate (EP) mediates protective effects after neuronal injury. Besides a direct conservation of damaged neurons, the modulation of indigenous glial cells has been suggested as one important mechanism for EP‐related neuroprotection. However, the specific contribution of glial cells is s...
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| Pubblicato in: | CNS Neurosci Ther |
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| Autori principali: | , , , , |
| Natura: | Artigo |
| Lingua: | Inglês |
| Pubblicazione: |
John Wiley and Sons Inc.
2017
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| Soggetti: | |
| Accesso online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6492706/ https://ncbi.nlm.nih.gov/pubmed/28836378 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/cns.12725 |
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