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Acquired BRAF rearrangements induce secondary resistance to EGFR therapy in EGFR-mutated lung cancers
INTRODUCTION: Multiple genetic mechanisms have been identified in EGFR-mutant lung cancers as mediators of acquired resistance (AR) to EGFR tyrosine kinase inhibitors (TKI) but many cases still lack a known mechanism. METHODS: To identify novel mechanisms of AR, we performed targeted large panel seq...
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| Pubblicato in: | J Thorac Oncol |
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| Autori principali: | , , , , , , , , , , , , , , , , |
| Natura: | Artigo |
| Lingua: | Inglês |
| Pubblicazione: |
2019
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| Soggetti: | |
| Accesso online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6486868/ https://ncbi.nlm.nih.gov/pubmed/30831205 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.jtho.2018.12.038 |
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