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CARDIAC-SPECIFIC DELETION OF GCN5L1 RESTRICTS RECOVERY FROM ISCHEMIA-REPERFUSION INJURY
GCN5L1 regulates mitochondrial protein acetylation, cellular bioenergetics, reactive oxygen species (ROS) generation, and organelle positioning in a number of diverse cell types. However, the functional role of GCN5L1 in the heart is currently unknown. As many of the factors regulated by GCN5L1 play...
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| Gepubliceerd in: | J Mol Cell Cardiol |
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| Hoofdauteurs: | , , , , , , , , , |
| Formaat: | Artigo |
| Taal: | Inglês |
| Gepubliceerd in: |
2019
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| Onderwerpen: | |
| Online toegang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6486843/ https://ncbi.nlm.nih.gov/pubmed/30776374 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.yjmcc.2019.02.009 |
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