Exogenous ubiquitin reduces inflammatory response and preserves myocardial function 3 days post-ischemia-reperfusion injury

β-Adrenergic receptor (β-AR) stimulation increases extracellular levels of ubiquitin (UB) in myocytes, and exogenous UB decreases β-AR-stimulated myocyte apoptosis and myocardial fibrosis. Here, we hypothesized that exogenous UB modulates the inflammatory response, thereby playing a protective role...

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Gorde:
Xehetasun bibliografikoak
Argitaratua izan da:Am J Physiol Heart Circ Physiol
Egile Nagusiak: Scofield, Stephanie L. C., Dalal, Suman, Lim, Kristina A., Thrasher, Patsy R., Daniels, Christopher R., Peterson, Jonathan M., Singh, Mahipal, Singh, Krishna
Formatua: Artigo
Hizkuntza:Inglês
Argitaratua: American Physiological Society 2019
Gaiak:
Research Article
Sarrera elektronikoa:https://ncbi.nlm.nih.gov/pmc/articles/PMC6459315/
https://ncbi.nlm.nih.gov/pubmed/30681370
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpheart.00654.2018
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