Ataxia telangiectasia-mutated kinase deficiency exacerbates left ventricular dysfunction and remodeling late after myocardial infarction

Ataxia telangiectasia-mutated kinase (ATM), a cell cycle checkpoint protein, is activated in response to DNA damage and oxidative stress. We have previously shown that ATM deficiency is associated with increased apoptosis and fibrosis and attenuation of cardiac dysfunction early (1–7 days) following...

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Udgivet i:Am J Physiol Heart Circ Physiol
Main Authors: Daniel, Laura L., Scofield, Stephanie L. C., Thrasher, Patsy, Dalal, Suman, Daniels, Christopher R., Foster, Cerrone R., Singh, Mahipal, Singh, Krishna
Format: Artigo
Sprog:Inglês
Udgivet: American Physiological Society 2016
Fag:
Integrative Cardiovascular Physiology and Pathophysiology
Online adgang:https://ncbi.nlm.nih.gov/pmc/articles/PMC5005283/
https://ncbi.nlm.nih.gov/pubmed/27288435
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpheart.00338.2016
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