Ataxia telangiectasia-mutated kinase deficiency exacerbates left ventricular dysfunction and remodeling late after myocardial infarction

Ataxia telangiectasia-mutated kinase (ATM), a cell cycle checkpoint protein, is activated in response to DNA damage and oxidative stress. We have previously shown that ATM deficiency is associated with increased apoptosis and fibrosis and attenuation of cardiac dysfunction early (1–7 days) following...

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Bibliografiset tiedot
Julkaisussa:Am J Physiol Heart Circ Physiol
Päätekijät: Daniel, Laura L., Scofield, Stephanie L. C., Thrasher, Patsy, Dalal, Suman, Daniels, Christopher R., Foster, Cerrone R., Singh, Mahipal, Singh, Krishna
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: American Physiological Society 2016
Aiheet:
Integrative Cardiovascular Physiology and Pathophysiology
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC5005283/
https://ncbi.nlm.nih.gov/pubmed/27288435
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpheart.00338.2016
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