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Alagebrium targets the miR-27b/TSP-1 signaling pathway to rescue N(ε)-carboxymethyl-lysine-induced endothelial dysfunction
N(ε)-carboxymethyl-lysine (CML), a major isoform of advanced glycation end products (AGEs), plays a crucial role in the functional damage of diabetes mellitus. However, it is not clear whether ALT-711 (alagebrium), an inhibitor of AGEs, is capable to rescue CML-induced poor angiogenesis, as well as...
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| Publicado no: | Am J Transl Res |
|---|---|
| Main Authors: | , , , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
e-Century Publishing Corporation
2019
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6456531/ https://ncbi.nlm.nih.gov/pubmed/30972183 |
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