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Disruption of the FasL/Fas axis protects against inflammation-derived tumorigenesis in chronic liver disease

Fas Ligand (FasL) and Fas (APO-1/CD95) are members of the TNFR superfamily and may trigger apoptosis. Here, we aimed to elucidate the functional role of Fas signaling in an experimental model of chronic liver disease, the hepatocyte-specific NEMO knockout (NEMO(Δhepa)) mice. We generated NEMO(Δhepa)...

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Detalhes bibliográficos
Publicado no:Cell Death Dis
Principais autores: Cubero, Francisco Javier, Woitok, Marius Maximilian, Zoubek, Miguel E., de Bruin, Alain, Hatting, Maximilian, Trautwein, Christian
Formato: Artigo
Idioma:Inglês
Publicado em: Nature Publishing Group UK 2019
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC6368573/
https://ncbi.nlm.nih.gov/pubmed/30737368
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s41419-019-1391-x
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