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Compensatory mechanisms for methylglyoxal detoxification in experimental & clinical diabetes
OBJECTIVES: The deficit of Glyoxalase I (Glo1) and the subsequent increase in methylglyoxal (MG) has been reported to be one the five mechanisms by which hyperglycemia causes diabetic late complications. Aldo-keto reductases (AKR) have been shown to metabolize MG; however, the relative contribution...
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| 出版年: | Mol Metab |
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| 主要な著者: | , , , , , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
Elsevier
2018
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6308908/ https://ncbi.nlm.nih.gov/pubmed/30287091 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.molmet.2018.09.005 |
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