Death-Domain-Receptor 3 Deletion Normalizes Inflammatory Gene Expression and Prevents Ileitis in Experimental Crohn’s Disease

Ítems similars

• Intestinal-Specific TNFα Overexpression Induces Crohn’s-Like Ileitis in Mice
  per: Bamias, Giorgos, et al.
  Publicat: (2013)
• Uncovering Pathogenic Mechanisms of Inflammatory Bowel Disease Using Mouse Models of Crohn’s Disease–Like Ileitis: What is the Right Model?
  per: Cominelli, Fabio, et al.
  Publicat: (2017)
• Neutralization of IL-1α ameliorates Crohn’s disease-like ileitis by functional alterations of the gut microbiome
  per: Menghini, Paola, et al.
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• The SAMP1/YitFc Mouse Strain: A Spontaneous Model of Crohn’s Disease-Like Ileitis
  per: Pizarro, Theresa T., et al.
  Publicat: (2011)
• Death Receptor 3 Signaling Controls the Balance between Regulatory and Effector Lymphocytes in SAMP1/YitFc Mice with Crohn’s Disease-Like Ileitis
  per: Li, Zhaodong, et al.
  Publicat: (2018)