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Notch signaling suppresses glucose metabolism in mesenchymal progenitors to restrict osteoblast differentiation
Notch signaling critically controls cell fate decisions in mammals, both during embryogenesis and in adults. In the skeleton, Notch suppresses osteoblast differentiation and sustains bone marrow mesenchymal progenitors during postnatal life. Stabilizing mutations of Notch2 cause Hajdu-Cheney syndrom...
Uloženo v:
| Vydáno v: | J Clin Invest |
|---|---|
| Hlavní autoři: | , |
| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
American Society for Clinical Investigation
2018
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6264656/ https://ncbi.nlm.nih.gov/pubmed/30284985 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1172/JCI96221 |
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