Melatonin protects endothelial progenitor cells against AGE-induced apoptosis via autophagy flux stimulation and promotes wound healing in diabetic mice

Wound healing is delayed in diabetic patients. Increased apoptosis and endothelial progenitor cell (EPC) dysfunction are implicated in delayed diabetic wound healing. Melatonin, a major secretory product of the pineal gland, promotes diabetic wound healing; however, its mechanism of action remains u...

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Detaylı Bibliyografya
Yayımlandı:Exp Mol Med
Asıl Yazarlar: Jin, Haiming, Zhang, Zengjie, Wang, Chengui, Tang, Qian, Wang, Jianle, Bai, Xueqin, Wang, Qingqing, Nisar, Majid, Tian, Naifeng, Wang, Quan, Mao, Cong, Zhang, Xiaolei, Wang, Xiangyang
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: Nature Publishing Group UK 2018
Konular:
Article
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC6249246/
https://ncbi.nlm.nih.gov/pubmed/30459300
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s12276-018-0177-z
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