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GSK3-β promotes calpain-1–mediated desmin filament depolymerization and myofibril loss in atrophy

Myofibril breakdown is a fundamental cause of muscle wasting and inevitable sequel of aging and disease. We demonstrated that myofibril loss requires depolymerization of the desmin cytoskeleton, which is activated by phosphorylation. Here, we developed a mass spectrometry–based kinase-trap assay and...

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Podrobná bibliografie
Vydáno v:J Cell Biol
Hlavní autoři: Aweida, Dina, Rudesky, Inga, Volodin, Alexandra, Shimko, Eitan, Cohen, Shenhav
Médium: Artigo
Jazyk:Inglês
Vydáno: Rockefeller University Press 2018
Témata:
On-line přístup:https://ncbi.nlm.nih.gov/pmc/articles/PMC6168250/
https://ncbi.nlm.nih.gov/pubmed/30061109
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1083/jcb.201802018
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