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Myofibril breakdown during atrophy is a delayed response requiring the transcription factor PAX4 and desmin depolymerization

A hallmark of muscle atrophy is the excessive degradation of myofibrillar proteins primarily by the ubiquitin proteasome system. In mice, during the rapid muscle atrophy induced by fasting, the desmin cytoskeleton and the attached Z-band–bound thin filaments are degraded after ubiquitination by the...

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Bibliografski detalji
Izdano u:Proc Natl Acad Sci U S A
Glavni autori: Volodin, Alexandra, Kosti, Idit, Goldberg, Alfred Lewis, Cohen, Shenhav
Format: Artigo
Jezik:Inglês
Izdano: National Academy of Sciences 2017
Teme:
Online pristup:https://ncbi.nlm.nih.gov/pmc/articles/PMC5338431/
https://ncbi.nlm.nih.gov/pubmed/28096335
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.1612988114
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