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GSK3-β promotes calpain-1–mediated desmin filament depolymerization and myofibril loss in atrophy

Myofibril breakdown is a fundamental cause of muscle wasting and inevitable sequel of aging and disease. We demonstrated that myofibril loss requires depolymerization of the desmin cytoskeleton, which is activated by phosphorylation. Here, we developed a mass spectrometry–based kinase-trap assay and...

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Dettagli Bibliografici
Pubblicato in:J Cell Biol
Autori principali: Aweida, Dina, Rudesky, Inga, Volodin, Alexandra, Shimko, Eitan, Cohen, Shenhav
Natura: Artigo
Lingua:Inglês
Pubblicazione: Rockefeller University Press 2018
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Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC6168250/
https://ncbi.nlm.nih.gov/pubmed/30061109
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1083/jcb.201802018
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