Defective mucin-type glycosylation on α-dystroglycan in COG-deficient cells increases its susceptibility to bacterial proteases

Deficiency in subunits of the conserved oligomeric Golgi (COG) complex results in pleiotropic defects in glycosylation and causes congenital disorders in humans. Insight regarding the functional consequences of this defective glycosylation and the identity of specific glycoproteins affected is lacki...

Deskribapen osoa

Gorde:
Xehetasun bibliografikoak
Argitaratua izan da:J Biol Chem
Egile Nagusiak: Yu, Seok-Ho, Zhao, Peng, Prabhakar, Pradeep K., Sun, Tiantian, Beedle, Aaron, Boons, Geert-Jan, Moremen, Kelley W., Wells, Lance, Steet, Richard
Formatua: Artigo
Hizkuntza:Inglês
Argitaratua: American Society for Biochemistry and Molecular Biology 2018
Gaiak:
Glycobiology and Extracellular Matrices
Sarrera elektronikoa:https://ncbi.nlm.nih.gov/pmc/articles/PMC6139567/
https://ncbi.nlm.nih.gov/pubmed/30049793
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.RA118.003014
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