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CD69 prevents PLZF(hi) innate precursors from prematurely exiting the thymus and aborting NKT2 cell differentiation
While CD69 may regulate thymocyte egress by inhibiting S1P(1) expression, CD69 expression is not thought to be required for normal thymocyte development. Here we show that CD69 is in fact specifically required for the differentiation of mature NKT2 cells, which do not themselves express CD69. Mechan...
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| Yayımlandı: | Nat Commun |
|---|---|
| Asıl Yazarlar: | , , , , , , , , , , , |
| Materyal Türü: | Artigo |
| Dil: | Inglês |
| Baskı/Yayın Bilgisi: |
Nature Publishing Group UK
2018
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| Konular: | |
| Online Erişim: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6138739/ https://ncbi.nlm.nih.gov/pubmed/30218105 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s41467-018-06283-1 |
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