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Overexpression of ZAC Impairs Glucose Stimulated Insulin Translation and Secretion in Clonal Pancreatic Beta-Cells
BACKGROUND: ZAC (Zinc finger protein that regulates Apoptosis and Cell-cycle arrest) is a candidate gene for transient neonatal diabetes mellitus (TNDM). This condition involves severe insulin deficiency at birth that reverses over weeks or months but may relapse with diabetes recurring in later lif...
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| Udgivet i: | Diabetes Metab Res Rev |
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| Main Authors: | , , , , |
| Format: | Artigo |
| Sprog: | Inglês |
| Udgivet: |
2012
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| Fag: | |
| Online adgang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6101213/ https://ncbi.nlm.nih.gov/pubmed/22865650 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1002/dmrr.2325 |
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