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Genetic Ablation of p16(INK4a) Does Not Protect against Cellular Senescence in Mouse Models of Chronic Obstructive Pulmonary Disease/Emphysema
Cigarette smoke (CS) affects DNA damage and cellular senescence signaling pathways in the pathogenesis of chronic obstructive pulmonary disease (COPD). p16(INK4a) (p16: a cyclin-dependent kinase inhibitor) is a key marker of cellular senescence, which is induced by CS in lung cells. It is thought th...
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| Publicat a: | Am J Respir Cell Mol Biol |
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| Autors principals: | , , , , |
| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
American Thoracic Society
2018
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6096345/ https://ncbi.nlm.nih.gov/pubmed/29447461 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1165/rcmb.2017-0390OC |
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