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Duplex signaling by CaM and Stac3 enhances Ca(V)1.1 function and provides insights into congenital myopathy

Ca(V)1.1 is essential for skeletal muscle excitation–contraction coupling. Its functional expression is tuned by numerous regulatory proteins, yet underlying modulatory mechanisms remain ambiguous as Ca(V)1.1 fails to function in heterologous systems. In this study, by dissecting channel trafficking...

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Dettagli Bibliografici
Pubblicato in:J Gen Physiol
Autori principali: Niu, Jacqueline, Yang, Wanjun, Yue, David T., Inoue, Takanari, Ben-Johny, Manu
Natura: Artigo
Lingua:Inglês
Pubblicazione: Rockefeller University Press 2018
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Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC6080896/
https://ncbi.nlm.nih.gov/pubmed/29950399
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1085/jgp.201812005
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