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Duplex signaling by CaM and Stac3 enhances Ca(V)1.1 function and provides insights into congenital myopathy
Ca(V)1.1 is essential for skeletal muscle excitation–contraction coupling. Its functional expression is tuned by numerous regulatory proteins, yet underlying modulatory mechanisms remain ambiguous as Ca(V)1.1 fails to function in heterologous systems. In this study, by dissecting channel trafficking...
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| Vydáno v: | J Gen Physiol |
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| Hlavní autoři: | , , , , |
| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
Rockefeller University Press
2018
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6080896/ https://ncbi.nlm.nih.gov/pubmed/29950399 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1085/jgp.201812005 |
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