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Ectopic overexpression of Kir6.1 in the mouse heart impacts on the life expectancy
We recently reported the reduced ATP-sensitive potassium (K(ATP)) channel activities in the transgenic mouse heart overexpressing the vascular type K(ATP) channel pore-forming subunit (Kir6.1). Although dysfunction of cardiac K(ATP) channel has been nominated as a cause of cardiomyopathy in human, t...
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| Gepubliceerd in: | Sci Rep |
|---|---|
| Hoofdauteurs: | , , , , , |
| Formaat: | Artigo |
| Taal: | Inglês |
| Gepubliceerd in: |
Nature Publishing Group UK
2018
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| Onderwerpen: | |
| Online toegang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6078942/ https://ncbi.nlm.nih.gov/pubmed/30082733 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s41598-018-30175-5 |
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