Protection conferred by myocardial ATP-sensitive K(+) channels in pressure overload-induced congestive heart failure revealed in KCNJ11 Kir6.2-null mutant

Ventricular load can precipitate development of the heart failure syndrome, yet the molecular components that control the cardiac adaptive response to imposed demand remain partly understood. Compromised ATP-sensitive K(+) (K(ATP)) channel function renders the heart vulnerable to stress, implicating...

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Detalles Bibliográficos
Main Authors: Yamada, Satsuki, Kane, Garvan C, Behfar, Atta, Liu, Xiao-Ke, Dyer, Roy B, Faustino, Randolph S, Miki, Takashi, Seino, Susumu, Terzic, Andre
Formato: Artigo
Idioma:Inglês
Publicado: Blackwell Science Inc 2006
Assuntos:
Integrative
Acceso en liña:https://ncbi.nlm.nih.gov/pmc/articles/PMC1890387/
https://ncbi.nlm.nih.gov/pubmed/17038430
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1113/jphysiol.2006.119511
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