ロード中...

Protection conferred by myocardial ATP-sensitive K(+) channels in pressure overload-induced congestive heart failure revealed in KCNJ11 Kir6.2-null mutant

Ventricular load can precipitate development of the heart failure syndrome, yet the molecular components that control the cardiac adaptive response to imposed demand remain partly understood. Compromised ATP-sensitive K(+) (K(ATP)) channel function renders the heart vulnerable to stress, implicating...

詳細記述

保存先:
書誌詳細
主要な著者: Yamada, Satsuki, Kane, Garvan C, Behfar, Atta, Liu, Xiao-Ke, Dyer, Roy B, Faustino, Randolph S, Miki, Takashi, Seino, Susumu, Terzic, Andre
フォーマット: Artigo
言語:Inglês
出版事項: Blackwell Science Inc 2006
主題:
オンライン・アクセス:https://ncbi.nlm.nih.gov/pmc/articles/PMC1890387/
https://ncbi.nlm.nih.gov/pubmed/17038430
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1113/jphysiol.2006.119511
タグ: タグ追加
タグなし, このレコードへの初めてのタグを付けませんか!