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Proteomic profiling of K(ATP) channel-deficient hypertensive heart maps risk for maladaptive cardiomyopathic outcome
KCNJ11 null mutants, lacking Kir6.2 ATP-sensitive K(+) (K(ATP)) channels, exhibit a marked susceptibility towards hypertension (HTN)-induced heart failure. To gain insight into the molecular alterations induced by knockout of this metabolic sensor under hemodynamic stress, wild-type (WT) and Kir6.2...
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| Main Authors: | , , , , , |
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| Format: | Artigo |
| Sprog: | Inglês |
| Udgivet: |
2009
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| Fag: | |
| Online adgang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2743411/ https://ncbi.nlm.nih.gov/pubmed/19253285 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1002/pmic.200800718 |
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