Lataa...
Proteomic profiling of K(ATP) channel-deficient hypertensive heart maps risk for maladaptive cardiomyopathic outcome
KCNJ11 null mutants, lacking Kir6.2 ATP-sensitive K(+) (K(ATP)) channels, exhibit a marked susceptibility towards hypertension (HTN)-induced heart failure. To gain insight into the molecular alterations induced by knockout of this metabolic sensor under hemodynamic stress, wild-type (WT) and Kir6.2...
Tallennettuna:
| Päätekijät: | , , , , , |
|---|---|
| Aineistotyyppi: | Artigo |
| Kieli: | Inglês |
| Julkaistu: |
2009
|
| Aiheet: | |
| Linkit: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2743411/ https://ncbi.nlm.nih.gov/pubmed/19253285 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1002/pmic.200800718 |
| Tagit: |
Lisää tagi
Ei tageja, Lisää ensimmäinen tagi!
|