GSK-3β governs inflammation-induced NFATc2 signaling hubs to promote pancreatic cancer progression

We aimed to investigate the mechanistic, functional and therapeutic role of GSK-3β in the regulation and activation of the pro-inflammatory oncogenic transcription factor NFATc2 in pancreatic cancer. Immunohistochemistry, quantitative real-time polymerase chain reaction, immunoblotting, immunofluore...

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Detalhes bibliográficos
Publicado no:Mol Cancer Ther
Main Authors: Baumgart, Sandra, Chen, Nai-ming, Zhang, Jin-San, Billadeau, Daniel D., Gaisina, Irina, Kozikowski, Alan P., Singh, Shiv, Fink, Daniel, Ströbel, Philipp, Klindt, Caroline, Zhang, Lizhi, Bamlet, William R., Koenig, Alexander, Hessmann, Elisabeth, Gress, Thomas, Ellenrieder, Volker, Neesse, Albrecht
Formato: Artigo
Idioma:Inglês
Publicado em: 2016
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Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC6071313/
https://ncbi.nlm.nih.gov/pubmed/26823495
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1158/1535-7163.MCT-15-0309
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