Atg9a deficiency causes axon-specific lesions including neuronal circuit dysgenesis

Conditional knockout mice for Atg9a, specifically in brain tissue, were generated to understand the roles of ATG9A in the neural tissue cells. The mice were born normally, but half of them died within one wk, and none lived beyond 4 wk of age. SQSTM1/p62 and NBR1, receptor proteins for selective aut...

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Detalhes bibliográficos
Publicado no:Autophagy
Main Authors: Yamaguchi, Junji, Suzuki, Chigure, Nanao, Tomohisa, Kakuta, Soichirou, Ozawa, Kentarou, Tanida, Isei, Saitoh, Tatsuya, Sunabori, Takehiko, Komatsu, Masaaki, Tanaka, Keiji, Aoki, Shigeki, Sakimura, Kenji, Uchiyama, Yasuo
Formato: Artigo
Idioma:Inglês
Publicado em: Taylor & Francis 2018
Assuntos:
Research Paper - Basic Science
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC6070006/
https://ncbi.nlm.nih.gov/pubmed/28513333
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1080/15548627.2017.1314897
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