CaMK4 compromises podocyte function in autoimmune and nonautoimmune kidney disease

Podocyte malfunction occurs in autoimmune and nonautoimmune kidney disease. Calcium signaling is essential for podocyte injury, but the role of Ca(2+)/calmodulin–dependent kinase (CaMK) signaling in podocytes has not been fully explored. We report that podocytes from patients with lupus nephritis an...

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Bibliografski detalji
Izdano u:J Clin Invest
Glavni autori: Maeda, Kayaho, Otomo, Kotaro, Yoshida, Nobuya, Abu-Asab, Mones S., Ichinose, Kunihiro, Nishino, Tomoya, Kono, Michihito, Ferretti, Andrew, Bhargava, Rhea, Maruyama, Shoichi, Bickerton, Sean, Fahmy, Tarek M., Tsokos, Maria G., Tsokos, George C.
Format: Artigo
Jezik:Inglês
Izdano: American Society for Clinical Investigation 2018
Teme:
Research Article
Online pristup:https://ncbi.nlm.nih.gov/pmc/articles/PMC6063476/
https://ncbi.nlm.nih.gov/pubmed/29985166
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1172/JCI99507
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