CaMK4 compromises podocyte function in autoimmune and nonautoimmune kidney disease

Podocyte malfunction occurs in autoimmune and nonautoimmune kidney disease. Calcium signaling is essential for podocyte injury, but the role of Ca(2+)/calmodulin–dependent kinase (CaMK) signaling in podocytes has not been fully explored. We report that podocytes from patients with lupus nephritis an...

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Bibliografiset tiedot
Julkaisussa:J Clin Invest
Päätekijät: Maeda, Kayaho, Otomo, Kotaro, Yoshida, Nobuya, Abu-Asab, Mones S., Ichinose, Kunihiro, Nishino, Tomoya, Kono, Michihito, Ferretti, Andrew, Bhargava, Rhea, Maruyama, Shoichi, Bickerton, Sean, Fahmy, Tarek M., Tsokos, Maria G., Tsokos, George C.
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: American Society for Clinical Investigation 2018
Aiheet:
Research Article
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC6063476/
https://ncbi.nlm.nih.gov/pubmed/29985166
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1172/JCI99507
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