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Tel1/ATM prevents degradation of replication forks that reverse after topoisomerase poisoning

In both yeast and mammals, the topoisomerase poison camptothecin (CPT) induces fork reversal, which has been proposed to stabilize replication forks, thus providing time for the repair of CPT‐induced lesions and supporting replication restart. We show that Tel1, the Saccharomyces cerevisiae ortholog...

Täydet tiedot

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Bibliografiset tiedot
Julkaisussa:EMBO Rep
Päätekijät: Menin, Luca, Ursich, Sebastian, Trovesi, Camilla, Zellweger, Ralph, Lopes, Massimo, Longhese, Maria Pia, Clerici, Michela
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: John Wiley and Sons Inc. 2018
Aiheet:
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC6030699/
https://ncbi.nlm.nih.gov/pubmed/29739811
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.15252/embr.201745535
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