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TGF-β signaling alters H4K20me3 status via miR-29 and contributes to cellular senescence and cardiac aging
Cellular senescence is a well-orchestrated programmed process involved in age-related pathologies, tumor suppression and embryonic development. TGF-β/Smad is one of the predominant pathways that regulate damage-induced and developmentally programmed senescence. Here we show that canonical TGF-β sign...
שמור ב:
| הוצא לאור ב: | Nat Commun |
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| Main Authors: | , , , , , , , , , , |
| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
Nature Publishing Group UK
2018
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6028646/ https://ncbi.nlm.nih.gov/pubmed/29967491 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s41467-018-04994-z |
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