Accumulation of cholera toxin and GM1 ganglioside in the early endosome of Niemann–Pick C1-deficient cells

Ítems similars

• Endoplasmic Reticulum-associated Degradation of Niemann-Pick C1: EVIDENCE FOR THE ROLE OF HEAT SHOCK PROTEINS AND IDENTIFICATION OF LYSINE RESIDUES THAT ACCEPT UBIQUITIN
  per: Nakasone, Naoe, et al.
  Publicat: (2014)
• Endosomal Accumulation of Toll-Like Receptor 4 Causes Constitutive Secretion of Cytokines and Activation of Signal Transducers and Activators of Transcription in Niemann–Pick Disease Type C (NPC) Fibroblasts: A Potential Basis for Glial Cell Activation in the NPC Brain
  per: Suzuki, Michitaka, et al.
  Publicat: (2007)
• Endosomal/Lysosomal Processing of Gangliosides Affects Neuronal Cholesterol Sequestration in Niemann-Pick Disease Type C
  per: Zhou, Sharon, et al.
  Publicat: (2011)
• GM1 ganglioside-independent intoxication by Cholera toxin
  per: Cervin, Jakob, et al.
  Publicat: (2018)
• The Wilson Disease Protein ATP7B Resides in the Late Endosomes with Rab7 and the Niemann-Pick C1 Protein
  per: Harada, Masaru, et al.
  Publicat: (2005)