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FAM35A associates with REV7 and modulates DNA damage responses of normal and BRCA1‐defective cells
To exploit vulnerabilities of tumors, it is urgent to identify associated defects in genome maintenance. One unsolved problem is the mechanism of regulation of DNA double‐strand break repair by REV7 in complex with 53BP1 and RIF1, and its influence on repair pathway choice between homologous recombi...
Tallennettuna:
| Julkaisussa: | EMBO J |
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| Päätekijät: | , , , , , , |
| Aineistotyyppi: | Artigo |
| Kieli: | Inglês |
| Julkaistu: |
John Wiley and Sons Inc.
2018
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| Aiheet: | |
| Linkit: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6003645/ https://ncbi.nlm.nih.gov/pubmed/29789392 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.15252/embj.201899543 |
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