Calcium/Calmodulin-Dependent Protein Kinase II Regulation of I(Ks) during Sustained Beta-Adrenergic Receptor Stimulation

BACKGROUND: Sustained β-adrenergic receptor (β-AR) stimulation causes pathophysiologic changes during heart failure (HF), including inhibition of the slow component of the delayed rectifier current, I(Ks). Aberrant calcium handling, including increased activation of calcium/calmodulin-dependent prot...

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Publicado en:Heart Rhythm
Autores principales: Shugg, Tyler, Johnson, Derrick E., Shao, Minghai, Lai, Xianyin, Witzmann, Frank, Cummins, Theodore R., Rubart-Von-der Lohe, Michael, Hudmon, Andy, Overholser, Brian R.
Formato: Artigo
Lenguaje:Inglês
Publicado: 2018
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Article
Acceso en línea:https://ncbi.nlm.nih.gov/pmc/articles/PMC5984714/
https://ncbi.nlm.nih.gov/pubmed/29410121
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.hrthm.2018.01.024
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