Brain mitochondrial iron accumulates in Huntington’s disease, mediates mitochondrial dysfunction, and can be removed pharmacologically

Lignende værker

• Novel proteomic changes in brain mitochondria provide insights into mitochondrial dysfunction in mouse models of Huntington’s disease
  af: Agrawal, Sonal, et al.
  Udgivet: (2019)
• Amyloid precursor protein haploinsufficiency preferentially mediates brain iron accumulation in mice transgenic for the Huntington’s disease mutation.
  af: Berggren, Kiersten, et al.
  Udgivet: (2017)
• Mitochondrial Dysfunction, Oxidative Stress and Neuroinflammation in Neurodegeneration with Brain Iron Accumulation (NBIA)
  af: Hinarejos, Isabel, et al.
  Udgivet: (2020)
• Neonatal Iron Supplementation Induces Striatal Atrophy in Female YAC128 Huntington’s Disease Mice
  af: Berggren, Kiersten L., et al.
  Udgivet: (2016)
• Mitochondrial dysfunction and defects in lipid homeostasis as therapeutic targets in neurodegeneration with brain iron accumulation
  af: Kinghorn, Kerri J., et al.
  Udgivet: (2016)