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Deletion of endoplasmic reticulum stress-responsive co-chaperone p58(IPK) protects mice from diet-induced steatohepatitis
Activation of the endoplasmic reticulum stress sensor PERK is a feature of nonalcoholic steatohepatitis (NASH), yet regulators of PERK signaling remain undefined in this context. P58(IPK) regulates PERK; however, its role in NASH has not been examined. Aim: the aim of this study was to assess the in...
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| Izdano u: | Hepatol Res |
|---|---|
| Glavni autori: | , , , , , , , |
| Format: | Artigo |
| Jezik: | Inglês |
| Izdano: |
2018
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| Teme: | |
| Online pristup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5932231/ https://ncbi.nlm.nih.gov/pubmed/29316085 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/hepr.13052 |
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