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TAK1 mediates excessive autophagy via p38 and ERK in cisplatin‐induced acute kidney injury

The ability of cisplatin (cis‐diamminedichloroplatinum II) toxicity to induce acute kidney injury (AKI) has attracted people's attention and concern for a long time, but its molecular mechanisms are still widely unknown. We found that the expression of transforming growth factor‐β (TGF‐β)‐activ...

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Опубликовано в: :J Cell Mol Med
Главные авторы: Zhou, Jun, Fan, Youling, Zhong, Jiying, Huang, Zhenxing, Huang, Teng, Lin, Sen, Chen, Hongtao
Формат: Artigo
Язык:Inglês
Опубликовано: John Wiley and Sons Inc. 2018
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Online-ссылка:https://ncbi.nlm.nih.gov/pmc/articles/PMC5908118/
https://ncbi.nlm.nih.gov/pubmed/29504713
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/jcmm.13585
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