TAK1 mediates excessive autophagy via p38 and ERK in cisplatin‐induced acute kidney injury

The ability of cisplatin (cis‐diamminedichloroplatinum II) toxicity to induce acute kidney injury (AKI) has attracted people's attention and concern for a long time, but its molecular mechanisms are still widely unknown. We found that the expression of transforming growth factor‐β (TGF‐β)‐activ...

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Bibliografiske detaljer
Udgivet i:J Cell Mol Med
Main Authors: Zhou, Jun, Fan, Youling, Zhong, Jiying, Huang, Zhenxing, Huang, Teng, Lin, Sen, Chen, Hongtao
Format: Artigo
Sprog:Inglês
Udgivet: John Wiley and Sons Inc. 2018
Fag:
Original Articles
Online adgang:https://ncbi.nlm.nih.gov/pmc/articles/PMC5908118/
https://ncbi.nlm.nih.gov/pubmed/29504713
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/jcmm.13585
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