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TAK1 mediates excessive autophagy via p38 and ERK in cisplatin‐induced acute kidney injury
The ability of cisplatin (cis‐diamminedichloroplatinum II) toxicity to induce acute kidney injury (AKI) has attracted people's attention and concern for a long time, but its molecular mechanisms are still widely unknown. We found that the expression of transforming growth factor‐β (TGF‐β)‐activ...
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| Publié dans: | J Cell Mol Med |
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| Auteurs principaux: | , , , , , , |
| Format: | Artigo |
| Langue: | Inglês |
| Publié: |
John Wiley and Sons Inc.
2018
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| Sujets: | |
| Accès en ligne: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5908118/ https://ncbi.nlm.nih.gov/pubmed/29504713 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/jcmm.13585 |
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