Class I HDACs control a JIP1-dependent pathway for kinesin-microtubule binding in cardiomyocytes

Class I histone deacetylase (HDAC) inhibitors block hypertrophy and fibrosis of the heart by suppressing pathological signaling and gene expression programs in cardiac myocytes and fibroblasts. The impact of HDAC inhibition in unstressed cardiac cells remains poorly understood. Here, we demonstrate...

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Bibliografske podrobnosti
izdano v:J Mol Cell Cardiol
Main Authors: Blakeslee, Weston W., Lin, Ying-Hsi, Stratton, Matthew S., Tatman, Philip D., Hu, Tianjing, Ferguson, Bradley S., McKinsey, Timothy A.
Format: Artigo
Jezik:Inglês
Izdano: 2017
Teme:
Article
Online dostop:https://ncbi.nlm.nih.gov/pmc/articles/PMC5845792/
https://ncbi.nlm.nih.gov/pubmed/28886967
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.yjmcc.2017.09.002
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