Class I HDACs control a JIP1-dependent pathway for kinesin-microtubule binding in cardiomyocytes

Class I histone deacetylase (HDAC) inhibitors block hypertrophy and fibrosis of the heart by suppressing pathological signaling and gene expression programs in cardiac myocytes and fibroblasts. The impact of HDAC inhibition in unstressed cardiac cells remains poorly understood. Here, we demonstrate...

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Detaylı Bibliyografya
Yayımlandı:J Mol Cell Cardiol
Asıl Yazarlar: Blakeslee, Weston W., Lin, Ying-Hsi, Stratton, Matthew S., Tatman, Philip D., Hu, Tianjing, Ferguson, Bradley S., McKinsey, Timothy A.
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: 2017
Konular:
Article
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC5845792/
https://ncbi.nlm.nih.gov/pubmed/28886967
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.yjmcc.2017.09.002
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