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Class I HDACs control a JIP1-dependent pathway for kinesin-microtubule binding in cardiomyocytes
Class I histone deacetylase (HDAC) inhibitors block hypertrophy and fibrosis of the heart by suppressing pathological signaling and gene expression programs in cardiac myocytes and fibroblasts. The impact of HDAC inhibition in unstressed cardiac cells remains poorly understood. Here, we demonstrate...
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| Publicado no: | J Mol Cell Cardiol |
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| Main Authors: | , , , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2017
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5845792/ https://ncbi.nlm.nih.gov/pubmed/28886967 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.yjmcc.2017.09.002 |
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