Chronic p38 mitogen-activated protein kinase inhibition improves vascular function and remodeling in angiotensin II-dependent hypertension

INTRODUCTION: An excess of angiotensin II (Ang II) causes hypertension and vascular injury. Activation of mitogen-activated protein kinase p38 (p38-MAPK) plays a substantial role in Ang II-dependent organ damage. Recently, we showed that p38-MAPK activation regulates the pressor response to Ang II....

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Detalhes bibliográficos
Publicado no:J Renin Angiotensin Aldosterone Syst
Main Authors: Potthoff, SA, Stamer, S, Grave, K, Königshausen, E, Sivritas, SH, Thieme, M, Mori, Y, Woznowski, M, Rump, LC, Stegbauer, J
Formato: Artigo
Idioma:Inglês
Publicado em: SAGE Publications 2016
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Original Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC5843849/
https://ncbi.nlm.nih.gov/pubmed/27407119
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1177/1470320316653284
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