Chronic treatment with angiotensin-(1-7) improves renal endothelial dysfunction in apolipoproteinE-deficient mice

BACKGROUND AND PURPOSE: ApolipoproteinE-deficient [apoE (−/−)] mice, a model of human atherosclerosis, develop endothelial dysfunction caused by decreased levels of nitric oxide (NO). The endogenous peptide, angiotensin-(1-7) [Ang-(1-7)], acting through its specific GPCR, the Mas receptor, has endot...

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Detalhes bibliográficos
Main Authors: Stegbauer, J, Potthoff, SA, Quack, I, Mergia, E, Clasen, T, Friedrich, S, Vonend, O, Woznowski, M, Königshausen, E, Sellin, L, Rump, LC
Formato: Artigo
Idioma:Inglês
Publicado em: Blackwell Science Inc 2011
Assuntos:
Research Papers
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC3130944/
https://ncbi.nlm.nih.gov/pubmed/21371005
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/j.1476-5381.2011.01295.x
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