Hyperoxia treatment of TREK-1/TREK-2/TRAAK-deficient mice is associated with a reduction in surfactant proteins

We previously proposed a role for the two-pore domain potassium (K2P) channel TREK-1 in hyperoxia (HO)-induced lung injury. To determine whether redundancy among the three TREK isoforms (TREK-1, TREK-2, and TRAAK) could protect from HO-induced injury, we now examined the effect of deletion of all th...

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Detalhes bibliográficos
Publicado no:Am J Physiol Lung Cell Mol Physiol
Main Authors: Schwingshackl, Andreas, Lopez, Benjamin, Teng, Bin, Luellen, Charlean, Lesage, Florian, Belperio, John, Olcese, Riccardo, Waters, Christopher M.
Formato: Artigo
Idioma:Inglês
Publicado em: American Physiological Society 2017
Assuntos:
Research Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC5814704/
https://ncbi.nlm.nih.gov/pubmed/28839101
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajplung.00121.2017
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