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Ratiometric sensing of BiP-client versus BiP levels by the unfolded protein response determines its signaling amplitude
Insufficient folding capacity of the endoplasmic reticulum (ER) activates the unfolded protein response (UPR) to restore homeostasis. Yet, how the UPR achieves ER homeostatic readjustment is poorly investigated, as in most studies the ER stress that is elicited cannot be overcome. Here we show that...
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| 出版年: | eLife |
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| 主要な著者: | , , , , , , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
eLife Sciences Publications, Ltd
2017
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5792092/ https://ncbi.nlm.nih.gov/pubmed/29251598 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.7554/eLife.27518 |
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