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Histone deacetylase inhibition attenuates hepatic steatosis in rats with experimental Cushing's syndrome
Cushing's syndrome (CS) is a collection of symptoms caused by prolonged exposure to excess cortisol. Chronically elevated glucocorticoid (GC) levels contribute to hepatic steatosis. We hypothesized that histone deacetylase inhibitors (HDACi) could attenuate hepatic steatosis through glucocortic...
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| Vydáno v: | Korean J Physiol Pharmacol |
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| Hlavní autoři: | , , , , , |
| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
The Korean Physiological Society and The Korean Society of Pharmacology
2018
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5746509/ https://ncbi.nlm.nih.gov/pubmed/29302209 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4196/kjpp.2018.22.1.23 |
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