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Salidroside attenuates hypoxia-induced pulmonary arterial smooth muscle cell proliferation and apoptosis resistance by upregulating autophagy through the AMPK-mTOR-ULK1 pathway

BACKGROUND: Recent studies have shown that both adenosine monophosphate activated protein kinase (AMPK) and the mammalian target of rapamycin (mTOR) are energy sensors and are related to autophagy. Our recent reports have shown that salidroside can exert protective effects against hypoxia-induced pu...

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محفوظ في:

التفاصيل البيبلوغرافية
الحاوية / القاعدة:	BMC Pulm Med
المؤلفون الرئيسيون:	Gui, Di, Cui, Zhimin, Zhang, Lin, Yu, Chang, Yao, Dan, Xu, Min, Chen, Mayun, Wu, Peiliang, Li, Guoping, Wang, Liangxing, Huang, Xiaoying
التنسيق:	Artigo
اللغة:	Inglês
منشور في:	BioMed Central 2017
الموضوعات:	Research Article
الوصول للمادة أونلاين:	https://ncbi.nlm.nih.gov/pmc/articles/PMC5726034/ https://ncbi.nlm.nih.gov/pubmed/29233105 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/s12890-017-0477-4
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Salidroside attenuates hypoxia-induced pulmonary arterial smooth muscle cell proliferation and apoptosis resistance by upregulating autophagy through the AMPK-mTOR-ULK1 pathway

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