ロード中...
Disruption of K(V)2.1 somato-dendritic clusters prevents the apoptogenic increase of potassium currents
As the predominant mediator of the delayed rectifier current, K(V)2.1 is an important regulator of neuronal excitability. K(V)2.1, however, also plays a well-established role in apoptotic cell death. Apoptogenic stimuli induce syntaxin-dependent trafficking of K(V)2.1, resulting in an augmented dela...
保存先:
| 出版年: | Neuroscience |
|---|---|
| 主要な著者: | , , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
2017
|
| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5709998/ https://ncbi.nlm.nih.gov/pubmed/28461216 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.neuroscience.2017.04.034 |
| タグ: |
タグ追加
タグなし, このレコードへの初めてのタグを付けませんか!
|