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Disruption of K(V)2.1 somato-dendritic clusters prevents the apoptogenic increase of potassium currents
As the predominant mediator of the delayed rectifier current, K(V)2.1 is an important regulator of neuronal excitability. K(V)2.1, however, also plays a well-established role in apoptotic cell death. Apoptogenic stimuli induce syntaxin-dependent trafficking of K(V)2.1, resulting in an augmented dela...
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| Publié dans: | Neuroscience |
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| Auteurs principaux: | , , , , , |
| Format: | Artigo |
| Langue: | Inglês |
| Publié: |
2017
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| Sujets: | |
| Accès en ligne: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5709998/ https://ncbi.nlm.nih.gov/pubmed/28461216 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.neuroscience.2017.04.034 |
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