טוען...
Deletion of Cdc42 in embryonic cardiomyocytes results in right ventricle hypoplasia
BACKGROUND: Cdc42 is a member of the Rho GTPase family and functions as a molecular switch in regulating cytoskeleton remodeling and cell polarity establishment. Inactivating Cdc42 in cardiomyocytes resulted in embryonic lethality with heart developmental defects, including ventricular septum defect...
שמור ב:
| הוצא לאור ב: | Clin Transl Med |
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| Main Authors: | , , , , , , , |
| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
Springer Berlin Heidelberg
2017
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5670094/ https://ncbi.nlm.nih.gov/pubmed/29101495 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/s40169-017-0171-4 |
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