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Uncoupling of endothelial nitric oxide synthase in cerebral vasculature of Tg2576 mice
In the present study we tested the hypothesis that reduced bioavailability of tetrahydrobiopterin (BH(4)) is a major mechanism responsible for pathogenesis of endothelial dysfunction in cerebral microvessels of transgenic mice expressing the Swedish double mutation of human amyloid precursor protein...
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| Gepubliceerd in: | J Neurochem |
|---|---|
| Hoofdauteurs: | , , , , , |
| Formaat: | Artigo |
| Taal: | Inglês |
| Gepubliceerd in: |
2015
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| Onderwerpen: | |
| Online toegang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5627976/ https://ncbi.nlm.nih.gov/pubmed/26111938 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/jnc.13205 |
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