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APOBEC3A and 3B Activities Render Cancer Cells Susceptible to ATR Inhibition

The apolipoprotein B mRNA editing enzyme catalytic polypeptide-like APOBEC3A and 3B have emerged as key mutation drivers in cancer. Here we show that APOBEC3A and 3B activities impose a unique type of replication stress by inducing abasic sites at replication forks. In contrast to cells under other...

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Detaylı Bibliyografya
Yayımlandı:Cancer Res
Asıl Yazarlar: Buisson, Rémi, Lawrence, Michael S., Benes, Cyril H., Zou, Lee
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: 2017
Konular:
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC5609510/
https://ncbi.nlm.nih.gov/pubmed/28698210
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1158/0008-5472.CAN-16-3389
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